How To Keep Your Penis Clean? पेनिस कैसे साफ़ करें? | Dr. Cuterus | Youth Ki Awaaz

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How To Keep Your Penis Clean? पेनिस कैसे साफ़ करें? | Dr. Cuterus | Youth Ki Awaaz

So you think you know how to keep it clean? Well you might get surprised. Watch this guide by @dr_cuterus to know some really important thing about penile hygiene.

P.S. It can help you avoid many serious health issues.

#MensHealth #drcuterus #PenileHygiene #reproductivehealth #sexualhealth

How To Keep Your Penis Clean | Dr. Cuterus | Youth Ki Awaaz

Youth Ki Awaaz is India’s largest and UN award-winning online platform for young people to express themselves on issues of critical importance.

Find exclusive youth views and video reports here, and videos that we love.

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Peninsular Mountains of India – Aravalli, Vindhya, Satpura, Western & Eastern Ghats | UPSC Geography

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In this video we will learn the about the mountains of Peninsular and Deccan plateau. They are – Aravalli, Vindhya, Satpura, Western Ghats & Eastern Ghats.

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The changing front lines of the Peninsular War in Spain and Portugal, between 1807 and 1814.

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Diabetic Ketoacidosis (DKA) Pathophysiology, Animation

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Diabetic ketoacidosis (one of the hyperglycemic crises), DKA, pathophysiology, causes, clinical presentation (signs and symptoms) and treatment. This video is available for instant download licensing here: https://www.alilamedicalmedia.com/-/galleries/narrated-videos-by-topics/diabetes/-/medias/bda71a7a-4598-4b1d-b298-ed06b3c54238-diabetic-ketoacidosis-dka-narrated-animation
Voice by: Penelope Hammet
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Diabetic ketoacidosis, DKA, is an ACUTE and potentially life-threatening complication of diabetes mellitus. DKA is commonly associated with type 1 but type 2 diabetics are also susceptible. DKA is caused by a critically LOW INSULIN level and is usually triggered when diabetic patients undergo further STRESS, such as infections, inadequate insulin administration, or cardiovascular diseases. It may also occur as the FIRST presentation of diabetes in people who did NOT know they had diabetes and therefore did NOT have insulin treatment.
Glucose is the MAJOR energy source of the body. It comes from digestion of carbohydrates and is carried by the bloodstream to various organs. Insulin is a hormone produced by beta-cells of the pancreas and is responsible for DRIVING glucose INTO cells. When insulin is DEFICIENT, glucose can NOT enter the cells; it stays in the blood, causing HIGH blood sugar levels while the cells are STARVED. In response to this metabolic starvation, the body INcreases the levels of counter-regulatory hormones. These hormones have 2 major effects that are responsible for clinical presentation of DKA:
– First, they produce MORE glucose in an attempt to supply energy to the cells. This is done by breaking down glycogen into glucose, and synthesizing glucose from NON-carbohydrate substrates such as proteins and lipids. However, as the cells CANNOT use glucose, this response ONLY results in MORE sugar in the blood. As blood sugar level EXCEEDS the ability of the kidneys to reabsorb, it overflows into urine, taking water and electrolytes along with it in a process known as OSMOTIC DIURESIS. This results in large volumes of urine, dehydration and excessive thirst.
– Second, they activate lipolysis and fatty acid metabolism for ALTERNATIVE fuel. In the liver, metabolism of fatty acids as an alternative energy source produces KETONE bodies. One of these is acetone, a volatile substance that gives DKA patient’s breath a characteristic SWEET smell. Ketone bodies, unlike fatty acids, can cross the blood-brain barrier and therefore can serve as fuel for the brain during glucose starvation. They are, however, ACIDIC, and when produced in LARGE amounts, overwhelm the buffering capacity of blood plasma, resulting in metabolic ACIDOSIS. As the body tries to reduce blood acidity by EXHALING MORE carbon dioxide, a deep and labored breathing, known as Kussmaul breathing may result. Another compensation mechanism for high acidity MOVES hydrogen ions INTO cells in exchange for potassium. This leads to INcreased potassium levels in the blood; but as potassium is constantly excreted in urine during osmotic diuresis, the overall potassium level in the body is eventually depleted. A blood test MAY indicate too much potassium, or hyperkalemia, but once INSULIN treatment starts, potassium moves BACK into cells and hypokalemia may result instead. For this reason, blood potassium level is monitored throughout treatment and potassium replacement is usually required together with intravenous fluid and insulin as primary treatment for DKA.

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Financial Focus: Impact of Marriage on Student Loans

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This video is for educational purposes. This session was presented on January 26, 2022, for RFUMS students.

Taxes as a married couple are already pretty complicated. But when you add in the fact that how you file your taxes as a married couple can greatly impact your student loan payments, it adds even more confusion. Check out our video where we cover the different ways couples can file their taxes, how it impacts your loan payment and determining the best option for your family.

*****Topics covered*****
– Tax Deductions, Tax Credits and Adjusted Gross Income
– Progressive Tax Brackets 101
– How To Know When It’s Worth It To File Taxes Separately

And much more!

Access our FREE webinar!
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*****Related Content*****

Revised Pay As You Earn For Married Couples

How Does The Senate Stimulus Package Affect My Student Loans?

*****About Ultra Borrowers Academy*****
Ultra Borrowers Academy exists to offer relief to people owing a mountain of student loan debt. Our course breaks down all of complexities of your federal and private student loans so that you can not only understand how your loans work, but also how to form a strategy so that student debt doesn’t prevent you from reaching your other financial goals. Join us as we tackle student loans together!

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Penile Pain – Why it Happens and How to Fix it | Pelvic Health and Physical Therapy

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Many men over the course of their lifetime will feel pain at the tip of their penis. Pelvic Floor expert Elizabeth Akincilar is here to talk about the nerves and muscles that are involved in this, why it occurs, and what you need to know to get better.

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About Pelvic Health and Rehabilitation Center:
PHRC is the leading physical therapy center for patients experiencing pelvic pain. At PHRC our advanced pelvic floor training allows us to integrate pelvic floor and pelvic girdle physiology to help the person function better as a whole. The benefit of our focus is that it has allowed us to establish a deep well of experience that we are able to draw from when treating this complex part of the body.

About Elizabeth Akincilar, MSPT
Liz received her Masters of Physical Therapy from the University of Miami Medical School. She is a Pennsylvania native but spent much of her early professional career in San Francisco. In 2015 Liz was excited to move back to the east coast to open PHRC’s first New England location. Now she is a fully acclimated New Englander who loves the four seasons. Liz’s happy place is in the kitchen cooking for friends and family or in the mountains with her partner and two rescue German Shepherds, Gunner and Jackson.
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#Rash on Penis? Common Causes of a Genital Rash and When to get Tested

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When you notice a rash on your penis you may immediately begin to worry. However, it’s important to know that most of the time, a penis rash may simply be a reaction to a new detergent or shaving product. Still, there are sexual causes of rashes on the penis which are worth knowing more about.

To navigate to specific points in this video, see below:
0:11 – What is a rash on the penis?
0:32 – What causes a rash on the penis?
2:41 – How can I get tested?

If it’s possible that your genital rash is a result of a sexually transmitted disease (#STD), it’s important to get checked. You can do this from the comfort of your own home with LetsGetChecked’s range of at-home STD tests which check for some of the most common STDs, including those which can cause a penis rash such as #herpes and #syphilis.

Home STD Test: https://www.letsgetchecked.com/home-std-test/
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Penile Discharge: Is It Normal?:
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Common STD Symptoms In Men: Should You Get Tested?:
https://www.letsgetchecked.com/articles/std-symptoms-in-men/

Can Herpes Go Away on Its Own?:
https://www.letsgetchecked.com/articles/can-herpes-go-away-on-its-own/

Four Steps to Prevent Getting or Spreading STDs:
https://www.letsgetchecked.com/articles/four-steps-to-prevent-getting-or-spreading-stds/

Are Viral STIs Curable?:
https://www.letsgetchecked.com/articles/are-viral-stis-curable/

Can Chlamydia Cause Long-Term Damage in Men?
https://www.letsgetchecked.com/articles/can-chlamydia-cause-long-term-damage-in-men/

LetsGetChecked is a virtual care company that allows customers to manage their #health from home, providing direct access to #telehealth services, #pharmacy, and laboratory tests with at-home sample collection kits for a wide range of health conditions including Sexual Health, #Cholesterol, #Diabetes, #Thyroid, Coronavirus (COVID-19), and more.

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Penile Carcinoma is a disease in which cancer cells form in the tissues of the penis. The first noticeable symptom of penile cancer is typically a lump, mass, or ulcer on the penis. It may look like a small, insignificant bump or a large, infected sore. Men who are uncircumcised are more likely to develop penile cancer.

Dr. Chandra Sekhar Patro, Consultant – Urologist at Narayana Multispeciality Hospital, Jessore Road, Kolkata explains the treatment, risk factors and details about Penile Carcinoma.

#NarayanaHealth #HealthForAll #AllForHealth #NHCares

For video consultation with the doctor, log on to https://bit.ly/2z7Y0Bd

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Chapters:
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1:00 – Risk Factor of Penis Cancer
1:15 – Phimosis
2:15 – Treatment
3:54 – Dr Points
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Hyperglycemia Symptoms and Treatment | High Blood Sugar Symptoms | Hyperglycemia vs Hypoglycemia

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Hyperglycemia is a condition that requires emergency treatment. Typically, patients with diabetes get hyperglycemia or high blood sugar. Lack of sufficient insulin in type 1 diabetes patients can lead to high blood sugar. Type 2 diabetes patients get it when their body’s insulin is ineffective.

There can be several causes of high blood sugar, including:
Skipping or forgetting to take insulin
Consuming more carbohydrates
Stress
Illness
Exercising less than normal
The dawn phenomenon (early morning surge in hormones)

If you suspect hyperglycemia, it is essential to seek treatment immediately since it can lead to life-threatening conditions. Early hyperglycemia symptoms are blood sugar higher than 180 mg/dL, increased thirst, frequent urination, headache, and fatigue. If not treated immediately, it can lead to nerve, blood vessel, and organ damage.

There are not too many differences present between hyperglycemia vs hypoglycemia. While hyperglycemia is high blood sugar (greater than 180 mg/dL), hypoglycemia is low blood sugar (less than 70 mg/dL). If not treated immediately, both of them can have severe consequences and may even lead to death.

How is #hyperglycemia treatment done? Can hyperglycemia cause ketoacidosis and HHNS, which are severe life-threatening conditions? We answer all your doubts in the video!

Like and share the video with your friends to let them know the dangers of high blood sugar! Subscribe to our channel for more such useful health information.

Chapters:
0:00 Intro
0:29 High Blood Sugar Causes
1:02 High Blood Sugar Symptoms
1:30 High Blood Sugar Complications
2:28 High Blood Sugar Treatment
2:50 Hyperglycemia vs Hypoglycemia

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Hyperglycemia - Causes, symptoms and treatment of hyperglycemia

Hyperglycemia is the term given to when your blood sugar levels are higher than 10 mmol/L. Blood sugar levels levels exceeding 7 mmol/L can damage internal organs, however, symptoms may not develop until blood glucose levels exceed 15 mmol/L so it’s worth knowing about the symptoms of hyperglycemia and how to recognise the symptoms.

Read more about hyperglycemia at: http://www.diabetes.co.uk/Diabetes-and-Hyperglycaemia.html
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How to Get Married

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How to Get Married

Getting married is a hugely significant and solemn step: for centuries, we did it under the guidance of religion, but nowadays, for many of us, faith no longer convinces. At the same time, we want to mark marriage with some kind of ritual and appropriate grandeur. That’s why the School of Life has redesigned the wedding ceremony, to bring it in line with modern ideals and the best insights of psychology.
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FURTHER READING

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The accident that changed the world – Allison Ramsey and Mary Staicu

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The accident that changed the world - Allison Ramsey and Mary Staicu

Learn how Alexander Fleming discovered penicillin, and how the antibiotic has changed medicine and the treatment of infections.

In 1928, scientist Alexander Fleming returned to his lab and found something unexpected: a colony of mold growing on a Petri dish he’d forgotten to place in his incubator. And around this colony of mold was a zone completely and surprisingly clear of bacteria. What was this mysterious phenomenon? Allison Ramsey and Mary Staicu detail the discovery of penicillin and how it transformed medicine.

Lesson by Allison Ramsey and Mary Staicu, directed by WOW-HOW Studio.

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Thank you so much to our patrons for your support! Without you this video would not be possible! Katrina Harding, Gerald Onyango, alessandra tasso, Astia Rizki Safitri, Côme Vincent, Antony Lee, Melissa Sorrells, Rakshit Kothari, Doreen Reynolds-Consolati, Simon Holst Ravn, Manognya Chakrapani, Ayala Ron, Samantha Chow, Philippe Spoden, Phyllis Dubrow, Michelle Stevens-Stanford, Cas Jamieson, Ophelia Gibson Best, Amber Wood, Paul Schneider, Jun Cai, Tim Robinson, Henrique ‘Sorín’ Cassús, cnorahs, Lyn-z Schulte, Elaine Fitzpatrick, Karthik Cherala, Clarence E. Harper Jr., Milad Mostafavi, Аркадий Скайуокер, Kiara Taylor, Louisa Lee, eden sher, Vignan Velivela, Sage Curie, Srikote Naewchampa, Tejas Dc, Khalifa Alhulail, Faiza Imtiaz, Martin Stephen, Jerome Froelich, Dan Paterniti, Jose Henrique Leopoldo e Silva, Mullaiarasu Sundaramurthy, Elnathan Joshua Bangayan, Caleb ross, Duo Xu, Quinn Shen, Marvin Vizuett and Sid.
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IMMUNE RESPONSE TO BACTERIAL INFECTION (Innate vs. Adaptive)

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A pricked finger means the immune system is hard at work. An important part of the innate immune system, the skin – has been breached, and bacteria are entering the body. The first immune cells they encounter are mast cells and dendritic cells. These cells can distinguish self from non-self thanks to the recognition of pathogen-associated molecular patterns, or PAMPs, which are molecules associated with pathogens. This recognition is not specific to any invader, but rather identifies a general attribute common to pathogens. This recognition is thanks to their pattern recognition receptors, or PRRs. The PAMPs they recognize can include bacterial lipopolysaccharides. Now that microbial components have been recognized, the body springs into action, and the inflammatory response is initiated.
The mast cells stay on the battlefield, releasing histamine and heparin. Histamine causes vasodilation of nearby blood vessels and heparin is an anticoagulant. The result is increased blood flow to the infected area, which allows more white blood cells to get there. The mast cells also release cytokines, which are cell signalling proteins that affect the behaviour of nearby cells. In this case, the cytokines are used to call macrophages and neutrophils to the area.
Neutrophils are the most abundant white blood cells. They release cytokines as well, amplifying the inflammatory response. They attack pathogens in three ways – phagocytosis (engulfing pathogens – and they can ingest up to 20 each), degranulation (release of soluble antimicrobials), and the release of neutrophil extracellular traps, or NETs. NETs are primarily composed of the neutrophils’ DNA and bind pathogens. This binding occurs thanks to positive charged proteins on the bacteria’s surface interacting with negatively charged chromatin fibers.
Dendritic cells engulf antigens – foreign substances that elicit an immune response – and break them up into smaller pieces called epitopes. Dendritic cells in the epithelial tissue move out of t he infected area and into the lymph nodes.
The innate immune system has non-specific means of intruder identification and resistance. However, when the dendritic cells enter the lymph nodes, they link the innate immune system to the adaptive immune system. The adaptive immune system consists of T cells and B cells, and brings in anti-pathogenic weaponry specific to the attacker.
T-cells are produced in the thymus, differentiating into four types: helper T-cells, cytotoxic T-cells, regulatory T-cells, or Tregs, and memory T-cells.
T-cells are specific to one antigen. After leaving the thymus, they circulate the body until an APC presents an antigen that matches their T-cell receptor, or TCR. Following this initial activation, the T-cell’s CD4 or CD8 molecule also binds the MHC of the APC, stabilizing the connection. Helper T-cells and cytotoxic T-cells also need secondary signals, as well as cytokines to become fully activated. Following these signals, the T-cell begins to divide rapidly and moves to the site of inflammation to fight the pathogen. At the infection site, mast cells, neutrophils, and epithelial cells can produce cytokines to induce further activation and proliferation of the T-cells.
Immature B-cells can be activated either by attaching to a free-floating antigen or thanks to helper T-cells or dendritic cells that present an epitope matching their B-cell receptors, or BCRs. BCRs consist of a membrane bound antibody, which is a large, Y-shaped protein that bind antigens, CD79A and CD79B. The B-cell receptor and antigen undergo cell-mediated endocytosis.
Recognition of an antigen stimulates B-cells to proliferate, and the activated B-cells undergo clonal expansion. As they proliferate, these many clones undergo somatic hypermutation. AID introduces point mutations into the clones. For some clones, this results in an increased affinity to the antigen, while for others, this means a decreased affinity. The antigen is proteolytically broken down and an epitope is then displayed on the B-cell’s surface, attached to an MHC class II protein. Before the B-cell can do anything, a helper T cell with a complementary TCR, and CD4+ glycoprotein must bind the antigen. The T helper cell then releases cytokines that let the B-cell take the next step. This is a safety mechanism to prevent accidental activation of the B-cells. The B-cells that have decreased affinity then undergo apoptosis, while the B-cells with increased affinity differentiate, becoming either a plasma cell, or a memory B-cell. The plasma cells produce antibodies matching their BCRs into the blood and lymph. Meanwhile, the memory B cells store antibodies in case of future reinfection.
When antibodies bind antigens, they label them for destruction by cells such as macrophages and neutrophils. B-cells mediate your humoral immune response, so called because it involves substances in your body fluids.
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What happens when you're infected with the COVID-19 coronavirus? | DW News

The vast majority of patients with the new coronavirus recover successfully without needing serious medical attention. So how many people recover – and what helps?
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