When you notice a rash on your penis you may immediately begin to worry. However, it’s important to know that most of the time, a penis rash may simply be a reaction to a new detergent or shaving product. Still, there are sexual causes of rashes on the penis which are worth knowing more about.

To navigate to specific points in this video, see below:
0:11 – What is a rash on the penis?
0:32 – What causes a rash on the penis?
2:41 – How can I get tested?

If it’s possible that your genital rash is a result of a sexually transmitted disease (#STD), it’s important to get checked. You can do this from the comfort of your own home with LetsGetChecked’s range of at-home STD tests which check for some of the most common STDs, including those which can cause a penis rash such as #herpes and #syphilis.

Home STD Test: https://www.letsgetchecked.com/home-std-test/
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Penile Discharge: Is It Normal?:
https://www.letsgetchecked.com/articles/penile-discharge-is-it-normal/

Common STD Symptoms In Men: Should You Get Tested?:
https://www.letsgetchecked.com/articles/std-symptoms-in-men/

Can Herpes Go Away on Its Own?:
https://www.letsgetchecked.com/articles/can-herpes-go-away-on-its-own/

Four Steps to Prevent Getting or Spreading STDs:
https://www.letsgetchecked.com/articles/four-steps-to-prevent-getting-or-spreading-stds/

Are Viral STIs Curable?:
https://www.letsgetchecked.com/articles/are-viral-stis-curable/

Can Chlamydia Cause Long-Term Damage in Men?
https://www.letsgetchecked.com/articles/can-chlamydia-cause-long-term-damage-in-men/

LetsGetChecked is a virtual care company that allows customers to manage their #health from home, providing direct access to #telehealth services, #pharmacy, and laboratory tests with at-home sample collection kits for a wide range of health conditions including Sexual Health, #Cholesterol, #Diabetes, #Thyroid, Coronavirus (COVID-19), and more.

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Penile Carcinoma is a disease in which cancer cells form in the tissues of the penis. The first noticeable symptom of penile cancer is typically a lump, mass, or ulcer on the penis. It may look like a small, insignificant bump or a large, infected sore. Men who are uncircumcised are more likely to develop penile cancer.

Dr. Chandra Sekhar Patro, Consultant – Urologist at Narayana Multispeciality Hospital, Jessore Road, Kolkata explains the treatment, risk factors and details about Penile Carcinoma.

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For video consultation with the doctor, log on to https://bit.ly/2z7Y0Bd

Visit our website https://www.narayanahealth.org to know more about Narayana Health and its facilities.

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Hyperglycemia is a condition that requires emergency treatment. Typically, patients with diabetes get hyperglycemia or high blood sugar. Lack of sufficient insulin in type 1 diabetes patients can lead to high blood sugar. Type 2 diabetes patients get it when their body’s insulin is ineffective.

There can be several causes of high blood sugar, including:
Skipping or forgetting to take insulin
Consuming more carbohydrates
Stress
Illness
Exercising less than normal
The dawn phenomenon (early morning surge in hormones)

If you suspect hyperglycemia, it is essential to seek treatment immediately since it can lead to life-threatening conditions. Early hyperglycemia symptoms are blood sugar higher than 180 mg/dL, increased thirst, frequent urination, headache, and fatigue. If not treated immediately, it can lead to nerve, blood vessel, and organ damage.

There are not too many differences present between hyperglycemia vs hypoglycemia. While hyperglycemia is high blood sugar (greater than 180 mg/dL), hypoglycemia is low blood sugar (less than 70 mg/dL). If not treated immediately, both of them can have severe consequences and may even lead to death.

How is #hyperglycemia treatment done? Can hyperglycemia cause ketoacidosis and HHNS, which are severe life-threatening conditions? We answer all your doubts in the video!

Like and share the video with your friends to let them know the dangers of high blood sugar! Subscribe to our channel for more such useful health information.

Chapters:
0:00 Intro
0:29 High Blood Sugar Causes
1:02 High Blood Sugar Symptoms
1:30 High Blood Sugar Complications
2:28 High Blood Sugar Treatment
2:50 Hyperglycemia vs Hypoglycemia

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Hyperglycemia is the term given to when your blood sugar levels are higher than 10 mmol/L. Blood sugar levels levels exceeding 7 mmol/L can damage internal organs, however, symptoms may not develop until blood glucose levels exceed 15 mmol/L so it’s worth knowing about the symptoms of hyperglycemia and how to recognise the symptoms.

Read more about hyperglycemia at: http://www.diabetes.co.uk/Diabetes-and-Hyperglycaemia.html
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Getting married is a hugely significant and solemn step: for centuries, we did it under the guidance of religion, but nowadays, for many of us, faith no longer convinces. At the same time, we want to mark marriage with some kind of ritual and appropriate grandeur. That’s why the School of Life has redesigned the wedding ceremony, to bring it in line with modern ideals and the best insights of psychology.
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Learn how Alexander Fleming discovered penicillin, and how the antibiotic has changed medicine and the treatment of infections.

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In 1928, scientist Alexander Fleming returned to his lab and found something unexpected: a colony of mold growing on a Petri dish he’d forgotten to place in his incubator. And around this colony of mold was a zone completely and surprisingly clear of bacteria. What was this mysterious phenomenon? Allison Ramsey and Mary Staicu detail the discovery of penicillin and how it transformed medicine.

Lesson by Allison Ramsey and Mary Staicu, directed by WOW-HOW Studio.

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A pricked finger means the immune system is hard at work. An important part of the innate immune system, the skin – has been breached, and bacteria are entering the body. The first immune cells they encounter are mast cells and dendritic cells. These cells can distinguish self from non-self thanks to the recognition of pathogen-associated molecular patterns, or PAMPs, which are molecules associated with pathogens. This recognition is not specific to any invader, but rather identifies a general attribute common to pathogens. This recognition is thanks to their pattern recognition receptors, or PRRs. The PAMPs they recognize can include bacterial lipopolysaccharides. Now that microbial components have been recognized, the body springs into action, and the inflammatory response is initiated.
The mast cells stay on the battlefield, releasing histamine and heparin. Histamine causes vasodilation of nearby blood vessels and heparin is an anticoagulant. The result is increased blood flow to the infected area, which allows more white blood cells to get there. The mast cells also release cytokines, which are cell signalling proteins that affect the behaviour of nearby cells. In this case, the cytokines are used to call macrophages and neutrophils to the area.
Neutrophils are the most abundant white blood cells. They release cytokines as well, amplifying the inflammatory response. They attack pathogens in three ways – phagocytosis (engulfing pathogens – and they can ingest up to 20 each), degranulation (release of soluble antimicrobials), and the release of neutrophil extracellular traps, or NETs. NETs are primarily composed of the neutrophils’ DNA and bind pathogens. This binding occurs thanks to positive charged proteins on the bacteria’s surface interacting with negatively charged chromatin fibers.
Dendritic cells engulf antigens – foreign substances that elicit an immune response – and break them up into smaller pieces called epitopes. Dendritic cells in the epithelial tissue move out of t he infected area and into the lymph nodes.
The innate immune system has non-specific means of intruder identification and resistance. However, when the dendritic cells enter the lymph nodes, they link the innate immune system to the adaptive immune system. The adaptive immune system consists of T cells and B cells, and brings in anti-pathogenic weaponry specific to the attacker.
T-cells are produced in the thymus, differentiating into four types: helper T-cells, cytotoxic T-cells, regulatory T-cells, or Tregs, and memory T-cells.
T-cells are specific to one antigen. After leaving the thymus, they circulate the body until an APC presents an antigen that matches their T-cell receptor, or TCR. Following this initial activation, the T-cell’s CD4 or CD8 molecule also binds the MHC of the APC, stabilizing the connection. Helper T-cells and cytotoxic T-cells also need secondary signals, as well as cytokines to become fully activated. Following these signals, the T-cell begins to divide rapidly and moves to the site of inflammation to fight the pathogen. At the infection site, mast cells, neutrophils, and epithelial cells can produce cytokines to induce further activation and proliferation of the T-cells.
Immature B-cells can be activated either by attaching to a free-floating antigen or thanks to helper T-cells or dendritic cells that present an epitope matching their B-cell receptors, or BCRs. BCRs consist of a membrane bound antibody, which is a large, Y-shaped protein that bind antigens, CD79A and CD79B. The B-cell receptor and antigen undergo cell-mediated endocytosis.
Recognition of an antigen stimulates B-cells to proliferate, and the activated B-cells undergo clonal expansion. As they proliferate, these many clones undergo somatic hypermutation. AID introduces point mutations into the clones. For some clones, this results in an increased affinity to the antigen, while for others, this means a decreased affinity. The antigen is proteolytically broken down and an epitope is then displayed on the B-cell’s surface, attached to an MHC class II protein. Before the B-cell can do anything, a helper T cell with a complementary TCR, and CD4+ glycoprotein must bind the antigen. The T helper cell then releases cytokines that let the B-cell take the next step. This is a safety mechanism to prevent accidental activation of the B-cells. The B-cells that have decreased affinity then undergo apoptosis, while the B-cells with increased affinity differentiate, becoming either a plasma cell, or a memory B-cell. The plasma cells produce antibodies matching their BCRs into the blood and lymph. Meanwhile, the memory B cells store antibodies in case of future reinfection.
When antibodies bind antigens, they label them for destruction by cells such as macrophages and neutrophils. B-cells mediate your humoral immune response, so called because it involves substances in your body fluids.
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The vast majority of patients with the new coronavirus recover successfully without needing serious medical attention. So how many people recover – and what helps?
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In this video Damien Fahy shares how you can potentially be missing out on £1,150.

Find out if you are eligible and how to claim here – https://moneytothemasses.com/tax/income-tax-2/marriage-tax-allowance-are-you-missing-out-on-1125-and-how-to-claim

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